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Franck Vazquez   Dr.  President, CEO or Director 
MDPI, Basel, Switzerland
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Franck Vazquez published an article in March 2018.
Research Keywords & Expertise
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Top co-authors See all
Hervé Vaucheret

104 shared publications

Institut Jean-Pierre Bourgin; UMR 1318; INRA; SPS Saclay Plant Sciences; Versailles France

Gaurav Sablok

21 shared publications

Department of Biodiversity and Molecular Ecology, Research and Innovation Centre, Fondazione Edmund Mach, Via E. Mach 1, San Michele all’Adige, 38010 Trento, Italy

Manfred Heinlein

16 shared publications

Université de Strasbourg; CNRS; IBMP UPR 2357; Strasbourg France

Mikhail M. Pooggin

16 shared publications

University of Basel, Hebelstarsse 1, Basel, Switzerland, 4058

Thomas Hohn

16 shared publications

Friedrich Miescher Institute, Basel, Switzerland

1 Following
Publication Record
Distribution of Articles published per year 
(2003 - 2018)
Total number of journals
published in
Publications See all
Article 8 Reads 3 Citations Crosstalk between PTGS and TGS pathways in natural antiviral immunity and disease recovery Camilla Julie Kørner, Nicolas Pitzalis, Eduardo José Peña, M... Published: 01 March 2018
Nature Plants, doi: 10.1038/s41477-018-0117-x
DOI See at publisher website
Article 8 Reads 0 Citations J—A Multidisciplinary Open Access Journal to Accelerate Scientific Communication Martyn Rittman, Franck Vazquez Published: 24 February 2018
J, doi: 10.3390/j1010001
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At its best, academic publishing facilitates the communication of the latest research results, accelerates sharing new and verified knowledge, and creates synergies between researchers in answering society’s most fundamental questions.
Article 6 Reads 0 Citations Welcome to the New Journal Biomimetics Franck Vazquez Published: 25 February 2016
Biomimetics, doi: 10.3390/biomimetics1010001
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Note: In lieu of an abstract, this is an excerpt from the first page.Excerpt Over geological time and through natural selection, living organisms have evolved specific organs, structures and materials to perform specific functions and allow them to survive and thrive in their environment.
Article 5 Reads 35 Citations Arabidopsis microRNA expression regulation in a wide range of abiotic stress responses Maria Barciszewska-Pacak, Katarzyna Knop, Dawid Bielewicz, F... Published: 04 June 2015
Frontiers in Plant Science, doi: 10.3389/fpls.2015.00410
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Arabidopsis microRNA expression regulation was studied in a wide array of abiotic stresses such as drought, heat, salinity, copper excess/deficiency, cadmium excess and sulphur deficiency. A home-built RT-qPCR mirEX platform for the amplification of 289 Arabidopsis microRNA transcripts was used to study their response to abiotic stresses. Small RNA sequencing, Northern hybridization and TaqMan® microRNA assays were performed to study the abundance of mature microRNAs. A broad response on the level of primary miRNAs (pri-miRNAs) was observed. However, stress response at the level of mature microRNAs was rather confined. The data presented show that in most instances, the level of a particular mature miRNA could not be predicted based on the level of its pri-miRNA. This points to an essential role of posttranscriptional regulation of microRNA expression. New Arabidopsis microRNAs responsive to abiotic stresses were discovered. Four microRNAs: miR319a/b, miR319b.2, and miR400 have been found to be responsive to several abiotic stresses and thus can be regarded as general stress-responsive microRNA species.
Article 0 Reads 0 Citations Welcome to the New Journal Non-Coding RNA! George A. Calin, Franck Vazquez Published: 21 November 2014
Non-Coding RNA, doi: 10.3390/ncrna1010001
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Note: In lieu of an abstract, this is an excerpt from the first page.Excerpt Dear colleagues and non-coding RNAs aficionados, you will probably ask 'why a new journal'?, these days when we are flooded not only by information of any type in any sector of our life, but also by so many new journals that come and disappear like comets in the summer sky! The answer is simple: because we believe that this field finally deserves to have a dedicated journal where its wide community will be able to communicate and exchange its latest findings in one centralized place. Moreover, with your outstanding contributions and by publishing papers that promulgate the 'thinking out of the box', we will be able to build a reputation for Non-Coding RNA to survive over the years to come.
Article 0 Reads 14 Citations miR393 Is Required for Production of Proper Auxin Signalling Outputs David Windels, Dawid Bielewicz, Miryam Ebneter, Artur Jarmol... Published: 24 April 2014
PLOS ONE, doi: 10.1371/journal.pone.0095972
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Auxins are crucial for plant growth and development. Auxin signalling primarily depends on four partially redundant F-box proteins of the TIR1/AFB2 Auxin Receptor (TAAR) clade to trigger the degradation of AUX/IAA transcriptional repressors. Auxin signalling is a balanced system which involves complex feedback regulations. miR393 regulation of TAAR genes is important for different developmental programs and for responses to environment. However, so far, the relevance of the two MIR393 genes for Arabidopsis leaf development and their significance for auxin signalling homeostasis have not been evaluated. First, our analyses of mir393a-1 and mir393b-1 mutants and of mir393ab double mutant show that the two genes have only partially redundant functions for leaf development. Expression analyses of typical auxin-induced reporter genes have shown that the loss of miR393 lead to several unanticipated changes in auxin signalling. The expression of DR5pro:GUS is decreased, the expression of primary AUX/IAA auxin-responsive genes is slightly increased and the degradation of the AXR3-NT:GUS reporter protein is delayed in mir393ab mutants. Additional analyses using synthetic auxin and auxin antagonists indicated that miR393 deficient mutants have higher levels of endogenous AUX/IAA proteins, which in turn create a competition for degradation. We propose that the counter-intuitive changes in the expression of AUX/IAA genes and in the accumulation of AUX/IAA proteins are explained by the intrinsic nature of AUX/IAA genes which are feedback regulated by the AUX/IAA proteins which they produce. Altogether our experiments provide an additional highlight of the complexity of auxin signaling homeostasis and show that miR393 is an important component of this homeostasis.